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<datestamp>2017-12-21</datestamp>
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<publisher>HAL CCSD</publisher>
<title lang=en>The CD95 signaling pathway: To not die and fly.</title>
<creator>Penna, Aubin</creator>
<creator>Khadra, Nadine</creator>
<creator>Tauzin, Sébastien</creator>
<creator>Vacher, Pierre</creator>
<creator>Legembre, Patrick</creator>
<contributor>Récepteur de mort et échappement tumoral ; Institut de recherche, santé, environnement et travail [Rennes] (Irset) ; Université d'Angers (UA) - Université des Antilles et de la Guyane (UAG) - Université de Rennes 1 (UR1) - École des Hautes Études en Santé Publique [EHESP] (EHESP) - Institut National de la Santé et de la Recherche Médicale (INSERM) - Structure Fédérative de Recherche en Biologie et Santé de Rennes ( Biosit : Biologie - Santé - Innovation Technologique ) - Université d'Angers (UA) - Université des Antilles et de la Guyane (UAG) - Université de Rennes 1 (UR1) - École des Hautes Études en Santé Publique [EHESP] (EHESP) - Institut National de la Santé et de la Recherche Médicale (INSERM) - Structure Fédérative de Recherche en Biologie et Santé de Rennes ( Biosit : Biologie - Santé - Innovation Technologique )</contributor>
<contributor>Validation et identification de nouvelles cibles en oncologie (VINCO) ; Université Bordeaux Segalen - Bordeaux 2 - Institut Bergonié - CRLCC Bordeaux - Institut National de la Santé et de la Recherche Médicale (INSERM)</contributor>
<contributor>Agence Nationale de la Recherche (ANR JC07_183182); INCa (projets libres recherche biomédicale); Cancéropole GO; Région Bretagne; Rennes Métropole; Université de Rennes-1; Ligue Contre le Cancer (Comités d'Ille-et-Vilaine/du Morbihan/des Côtes d'Armor/du Maine et Loire/des Landes)</contributor>
<description>International audience</description>
<source>ISSN: 1942-0889</source>
<source>Communicative and Integrative Biology</source>
<publisher>Taylor & Francis Open</publisher>
<identifier>hal-00873761</identifier>
<identifier>https://hal.archives-ouvertes.fr/hal-00873761</identifier>
<identifier>https://hal.archives-ouvertes.fr/hal-00873761/document</identifier>
<identifier>https://hal.archives-ouvertes.fr/hal-00873761/file/The_CD95_signaling_pathway-accepted.pdf</identifier>
<source>https://hal.archives-ouvertes.fr/hal-00873761</source>
<source>Communicative and Integrative Biology, Taylor & Francis Open, 2012, 5 (2), pp.190-2. 〈10.4161/cib.18888〉</source>
<identifier>DOI : 10.4161/cib.18888</identifier>
<relation>info:eu-repo/semantics/altIdentifier/doi/10.4161/cib.18888</relation>
<identifier>PUBMED : 22808330</identifier>
<relation>info:eu-repo/semantics/altIdentifier/pmid/22808330</relation>
<language>en</language>
<subject lang=en>Apoptosis</subject>
<subject lang=en>calcium</subject>
<subject lang=en>Fas</subject>
<subject lang=en>FasL</subject>
<subject lang=en>lymphocyte</subject>
<subject lang=en>migration</subject>
<subject lang=en>Orai1</subject>
<subject lang=en>PI3K</subject>
<subject lang=en>TNF</subject>
<subject>[SDV.BC] Life Sciences [q-bio]/Cellular Biology</subject>
<subject>[SDV.CAN] Life Sciences [q-bio]/Cancer</subject>
<type>info:eu-repo/semantics/article</type>
<type>Journal articles</type>
<description lang=en>Our recent findings indicate that cells exposed to transmembrane (m-CD95L) or metalloprotease-cleaved CD95L (cl-CD95L) undergo a localized Ca(2+)entry that not only inhibits the initial steps of the CD95-mediated apoptotic signal but also promotes cell motility. Based on recent findings published on the non-apoptotic signals induced by CD95, we discuss how m-CD95L and cl-CD95L diverging by their stoichiometry could both contribute to the immune response by first recruiting activated T lymphocytes in the inflamed area and later by eliminating infected and transformed cells.</description>
<date>2012-03-01</date>
</dc>
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